The Pattern of p53 Mutations Caused by PAH o-Quinones is Driven by 8-oxo-dGuo Formation while the Spectrum of Mutations is Determined by Biological Selection for Dominance | Chemical Research in Toxicology
Polycyclic Aromatic Hydrocarbon (PAH) o-Quinones Produced by the Aldo-Keto-Reductases (AKRs) Generate Abasic Sites, Oxidized Pyrimidines, and 8-Oxo-dGuo via Reactive Oxygen Species | Chemical Research in Toxicology
Formation of 8-oxo-7,8-dihydro-2'-deoxyguanosine (8-oxo-dGuo) by PAH o-quinones: involvement of reactive oxygen species and copper(II)/copper(I) redox cycling. - Abstract - Europe PMC
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Polycyclic Aromatic Hydrocarbon (PAH) o-Quinones Produced by the Aldo-Keto-Reductases (AKRs) Generate Abasic Sites, Oxidized Pyrimidines, and 8-Oxo-dGuo via Reactive Oxygen Species | Chemical Research in Toxicology
Low-level exposure to polycyclic aromatic hydrocarbons is associated with reduced lung function among Swedish young adults - ScienceDirect
Frontiers | Acquired disorders of mitochondrial metabolism and dynamics in pulmonary arterial hypertension
Beyond the obvious: Environmental health implications of polar polycyclic aromatic hydrocarbons - ScienceDirect
Polycyclic aromatic hydrocarbon exposure, oxidative potential in dust, and their relationships to oxidative stress in human body: A case study in the indoor environment of Guangzhou, South China - ScienceDirect
Biomarkers of PAH exposure and associated disease conditions in humans. | Download Table
Emerging Concepts in the Molecular Basis of Pulmonary Arterial Hypertension | Circulation
Pharmacology of Pulmonary Arterial Hypertension: An Overview of Current and Emerging Therapies | ACS Pharmacology & Translational Science
PDF) Correction to The Pattern of p53 Mutations Caused by PAH o -Quinones is Driven by 8-Oxo-dGuo Formation while the Spectrum of Mutations is Determined by Biological Selection for Dominance | Ian
Formation of 8-Oxo-7,8-dihydro-2'-deoxyguanosine (8-Oxo-dGuo) by PAH o-Quinones: Involvement of Reactive Oxygen Species and Copper(II)/Copper(I) Redox Cycling | Chemical Research in Toxicology
Frontiers | Glutaminolysis: A Driver of Vascular and Cardiac Remodeling in Pulmonary Arterial Hypertension
Covalent and oxidative DNA lesions caused by PAH o-quinones. | Download Scientific Diagram
Covalent and oxidative DNA lesions caused by PAH o-quinones. | Download Scientific Diagram
PDF) Oxidative damage mediates the association between polycyclic aromatic hydrocarbon exposure and lung function
Metabolomics of exercise pulmonary hypertension are intermediate between controls and patients with pulmonary arterial hypertension - Jason L. Sanders, Yuchi Han, Mariana F. Urbina, David M. Systrom, Aaron B. Waxman, 2019
Toxics | Free Full-Text | Urinary Polycyclic Aromatic Hydrocarbon Metabolites Are Associated with Biomarkers of Chronic Endocrine Stress, Oxidative Stress, and Inflammation in Adolescents: FLEHS-4 (2016–2020)
Metabolomics-based mechanism exploration of pulmonary arterial hypertension pathogenesis: novel lessons from explanted human lungs | Hypertension Research
IJERPH | Free Full-Text | Assessing Approaches of Human Inhalation Exposure to Polycyclic Aromatic Hydrocarbons: A Review
Formation of 8-Oxo-7,8-dihydro-2'-deoxyguanosine (8-Oxo-dGuo) by PAH o-Quinones: Involvement of Reactive Oxygen Species and Copper(II)/Copper(I) Redox Cycling | Chemical Research in Toxicology
